Seracare心肌磷脂IgM（Cardiolipin IgM）
| 型 号： | 美国 |
| 报 价： |  |
美国SeracareSeracare心肌磷脂IgM（Cardiolipin IgM） 阳性质控品对照品 需要了解更多Seracare产品可以咨询我们，本产品由广州健仑生物科技有限公司提供
- 产品描述
美国Seracare心肌磷脂IgM（Cardiolipin IgM）
广州健仑生物科技有限公司
广州健仑长期供应各种生物原料，主要代理品牌：美国Seracare、西班牙Certest、美国Fuller等等。
主要产品包括各种标准品、阳性对照品、单克隆抗原抗体。
其中常见的有：弓形虫病、西尼罗河病毒、类风湿因子、疟疾、麻疹、莱姆病、百日咳杆菌、大肠杆菌、鼠伤寒沙门氏菌、李斯特菌等阳性对照品。
美国Seracare心肌磷脂IgM（Cardiolipin IgM）
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【Seracare产品介绍】
编号 | 英文名称 | 中文名称 |
JL-FA-01 | Amebiasis (AME) | 阿米巴病 |
JL-FA-02 | Allergens, Rast scores | 过敏原，放射性过敏原吸收实验。指对特定的人群引起免疫反应或者过敏反应的食品中的蛋白质 |
JL-FA-03 | Allergens, Rast scores negative | 过敏原，放射性过敏原吸收实验阴性 |
JL-FA-04 | Anti-cyclic citrullinated peptide Antibody (CCP) Arthritis | 抗环瓜氨酸肽抗体 |
JL-FA-05 | ASCA Saccharomyces Cerevi | 人抗酿酒酵母抗体（ASCA） |
JL-FA-06 | Aspergillis | 麴菌病 |
JL-FA-07 | Beta 2 Glycoprotein | β2糖蛋白 |
JL-FA-08 | Beta 2 Glycoprotein IgM | β2糖蛋白 IGM |
JL-FA-09 | Bordela Pertussis | 百日咳杆菌 |
JL-FA-10 | Bordela Pertussis IgM | 百日咳杆菌 IGM |
JL-FA-11 | C-ANCA | C-抗中性粒细胞胞浆抗体（ANCA) |
JL-FA-12 | Cardiolipin | 心肌磷脂 |
JL-FA-13 | Cardiolipin IgA | 心肌磷脂 IGA |
JL-FA-14 | Cardiolipin IgG | 心肌磷脂 IGG |
JL-FA-15 | Cardiolipin IgM | 心肌磷脂 IGM |
JL-FA-16 | Cerebral Spinal Fluid | 脑脊髓液 |
JL-FA-17 | Chagas | 恰加斯病/南美锥虫 |
JL-FA-18 | Chlamydia | 衣原体 |
JL-FA-19 | Chlamydia IgA | 衣原体IGA |
JL-FA-20 | Chlamydia IgG | 衣原体IGG |
JL-FA-21 | Chlamydia IgM | 衣原体IGM |
JL-FA-22 | Chlamydia Neg | 衣原体阴性 |
JL-FA-23 | Clotting Factor C3 | 凝固因子C3 |
JL-FA-24 | Clotting Factor C4 | 凝固因子C4 |
JL-FA-25 | Coccidiodes | 球孢菌 |
JL-FA-26 | Cytomegalovirus (CMV) Neg | 巨细胞病毒抗体阴性 |
JL-FA-27 | CMV IgG | 巨细胞病毒 IGG阳性 |
JL-FA-28 | CMV IgM VCA | 巨细胞病毒 IGM 阳性 |
JL-FA-29 | C-Reactive Protein (CRP) | C-反应蛋白质 |
JL-FA-30 | Dengue Fever | 登革热 |
JL-FA-31 | Dengue Fever IgM | 登革热 IGM |
JL-FA-32 | DS (Double Stranded) DNA | 双链脱氧核糖核酸 |
JL-FA-33 | EBNA (Epstein-Barr nuclear antigen) IgG | EB病毒核抗原 IGG |
JL-FA-34 | EBNA (Epstein-Barr nuclear antigen) IgM | EB病毒核抗原 IGM |
JL-FA-35 | Epstein Barr Virus (EBV) Negative Plasma | EB病毒阴性血浆 |
JL-FA-36 | Epstein Barr Virus (EBV) EA IgM | EB病毒早期抗原 IGM |
JL-FA-37 | Epstein Barr Virus (EBV) VCA IgM | EB病毒壳蛋白 IGM |
JL-FA-38 | Epstein Barr Virus (EBV) EA IgG | EB病毒早期抗原 IGG |
JL-FA-39 | EMA (Endomysial Antibodies) | 肌内膜 |
JL-FA-40 | Gliadin | 麸蛋白,麦醇溶蛋白,麦胶蛋白 |
JL-FA-41 | Gliadin IgG | 麦醇溶蛋白 IGG |
JL-FA-42 | Gliadin IgA | 麦醇溶蛋白 IGA |
JL-FA-43 | Glomerular Basement Membrane (GBMA) | 肾小球基底膜病 |
JL-FA-44 | Helicobacter pylori IgA | 幽门螺旋杆菌IGA |
JL-FA-45 | Helicobacter pylori IgG | 幽门螺旋杆菌IGG |
JL-FA-46 | Helicobacter pylori IgM | 幽门螺旋杆菌IGM |
JL-FA-47 | Helicobacter pylori Negative | 幽门螺旋杆菌阴性 |
JL-FA-48 | Helicobacter pylori Positive Plasma | 幽门螺旋杆菌阴性血浆 |
JL-FA-49 | Hepatitis A Virus (HAV) Pos. Plasma | 甲型肝炎病毒阳性血浆 |
JL-FA-50 | Hepatitis A Virus (HAV) IgM | 甲型肝炎病毒IGM |
JL-FA-51 | Hepatitis B Core (HBc) IgG | 乙型肝炎病毒核心 IGG |
JL-FA-52 | Hepatitis B Core (HBc) IgM | 乙型肝炎病毒核心 IGM |
JL-FA-53 | Anti Hbe (Antibody to HBV antigen) | 乙肝抗体 |
JL-FA-54 | Hepatitis Delta Virus | 丁型肝炎病毒 |
JL-FA-55 | HBeAg (HBV e antigen) | 乙肝 E抗原 |
JL-FA-56 | anti-HBs (HBV surface antibody) | 乙肝表面抗体 |
JL-FA-57 | Hepatitis B (HBsAg) "Chronic" | 乙型肝炎（乙肝表面抗原）“慢性病 |
JL-FA-58 | HBsAg (HBV surface antigen) Serum | 乙肝表面抗原血清 |
JL-FA-59 | HBsAg (AD) | 乙肝表面抗原（AD） |
JL-FA-60 | HBsAg (AY) | 乙肝表面抗原（AY） |
JL-FA-61 | HBV Positive Plasma | 乙肝阳性血浆 |
JL-FA-62 | HBV DNA Plasma | 乙肝DNA血浆 |
JL-FA-63 | HBV DNA Serum | 乙肝DNA血清 |
JL-FA-64 | HBV DNA type A | A型 乙肝DNA |
JL-FA-65 | HBV DNA type B | B型 乙肝DNA |
JL-FA-66 | HBV DNA type C | C型 乙肝DNA |
JL-FA-67 | HBV DNA type D | D型 乙肝DNA |
JL-FA-68 | HBV DNA type E | E型 乙肝DNA |
JL-FA-69 | HBV DNA type F | F型 乙肝DNA |
JL-FA-70 | HBV Antibody HCV Antibody Plasma CO-INFECTED | 乙肝和丙肝联合感染血浆 |
JL-FA-71 | HCV (Hepatitis C Virus) Antibody | 丙型肝炎抗体 |
JL-FA-72 | HCV Core Antigen Positive | 丙肝核心抗原 阳性 |
JL-FA-73 | HCV RNA PLASMA Genotype 1 | 基因1型丙肝RNA 血浆 |
JL-FA-74 | HCV RNA PLASMA Genotype 2 | 基因2型丙肝RNA 血浆 |
JL-FA-75 | HCV RNA PLASMA Genotype 3 | 基因3型丙肝RNA 血浆 |
JL-FA-76 | HCV RNA PLASMA Genotype 4 | 基因4型丙肝RNA 血浆 |
JL-FA-77 | HCV RNA PLASMA Genotype 5 | 基因5型丙肝RNA 血浆 |
JL-FA-78 | HCV RNA PLASMA Genotype 6 | 基因6型丙肝RNA 血浆 |
JL-FA-79 | HCV Riba single band | 丙肝免疫印迹单波段 |
JL-FA-80 | HCV RIBA Pos. (multiple bands) | 丙肝免疫印迹阳性多波段 |
JL-FA-81 | HCV Negative | 丙肝阴性 |
JL-FA-82 | HCV RNA Pos (quantitative) | 丙肝RNA阳性（定量） |
JL-FA-83 | Hepatitis E | 戊型肝炎 |
JL-FA-84 | Herpes Simplex Virus (HSV)1/2 Positive Plasma | 单纯性疱疹病毒1/2阳性血浆 |
JL-FA-85 | Herpes Simplex Virus (HSV) 1 Negative Plasma | 单纯性疱疹病毒1 阴性血浆 |
JL-FA-86 | Herpes Simplex Virus (HSV) 1 IgG | 单纯性疱疹病毒1 IGG |
JL-FA-87 | Herpes Simplex Virus (HSV 1) IgM | 单纯性疱疹病毒1 IGM |
JL-FA-88 | Herpes Simplex Virus (HSV) 2 IgG | 单纯性疱疹病毒2 IGG |
JL-FA-89 | Herpes Simplex Virus (HSV) 2 IgM | 单纯性疱疹病毒2 IGG |
JL-FA-90 | Histone | 组蛋白 |
JL-FA-91 | Human Anti Mouse Ab (HAMA) | 人抗鼠抗体 |
JL-FA-92 | Human immunodeficiency virus (HIV) 1 Neg | HIV I 阴性 |
JL-FA-93 | anti Human immunodeficiency virus (HIV) 1 Plasma | 抗HIV I 血浆 |
JL-FA-94 | anti Human immunodeficiency virus (HIV) 1 Serum | 抗HIV I 血清 |
JL-FA-95 | anti Human immunodeficiency virus (HIV) 2 Western Blot Tested | 抗HIV 2 免疫印迹 |
JL-FA-96 | anti Human immunodeficiency virus (HIV) 1/2 2 HIV (+) | 抗HIV 1/2 2 HIV阳性 |
JL-FA-97 | Human immunodeficiency virus (HIV) Ag | HIV抗原 |
JL-FA-98 | HIV RNA (quantitative) Plasma | HIV RNA 定量血浆 |
JL-FA-99 | HIV RNA (quantitative) Serum | HIV RNA 定量血清 |
JL-FA-100 | HIV1 Subtype A | HIV1 亚型A |
JL-FA-101 | HIV1 Subtype B | HIV1 亚型B |
JL-FA-102 | HIV1 Subtype C | HIV1 亚型C |
JL-FA-103 | HIV1 Subtype D | HIV1 亚型D |
JL-FA-104 | HIV1 Subtype E | HIV1 亚型E |
JL-FA-105 | HIV1 Subtype F | HIV1 亚型F |
JL-FA-106 | HIV1 Subtype G | HIV1 亚型G |
JL-FA-107 | HIV1 Subtype H | HIV1 亚型H |
JL-FA-108 | HIV1 Subtype J | HIV1 亚型J |
JL-FA-109 | HIV1 Subtype K | HIV1 亚型K |
JL-FA-110 | HIV1 Group O | HIV1 亚型O |
JL-FA-111 | Human immunodeficiency virus (HIV) 2 Antibody Plasma | HIV 2 抗体血浆 |
JL-FA-112 | Human immunodeficiency virus (HIV) 2 Antibody Serum | HIV 2 抗体血清 |
JL-FA-113 | HPV (Human Papiloma Virus) Negative | 人乳状瘤病毒HPV阴性 |
JL-FA-114 | HPV (Human Papiloma Virus) Positive | 人乳状瘤病毒HPV阳性 |
JL-FA-115 | Human immunodeficiency virus (HIV) Antibody HCV Antibody Plasma COINFECTED | HIV 抗体 HCV |
JL-FA-116 | Human T-cell Lymphotropic Virus (HTLV) I/II | 人嗜T淋巴细胞病毒(HTLV) I/II |
JL-FA-117 | Human T-cell Lymphotropic Virus (HTLV) I | 人嗜T淋巴细胞病毒(HTLV) I |
JL-FA-118 | Human T-cell Lymphotropic Virus (HTLV) II | 人嗜T淋巴细胞病毒(HTLV) II |
JL-FA-119 | Jo-1 | 多发性肌炎抗原JO-1 |
JL-FA-120 | IgE < 5,000 Ku/L | IgE < 5,000 Ku/L |
JL-FA-121 | Legionella | 军团杆菌属 |
JL-FA-122 | Leptospira | 军团杆菌属 |
JL-FA-123 | Lyme Disease | 莱姆(氏)病:蜱传播的全身性疾病,常在夏季发生 |
JL-FA-124 | Lyme IgG | 莱姆(氏)病 IGG |
JL-FA-125 | Lyme IgM | 莱姆(氏)病 IGM |
JL-FA-126 | Lyme Disease Neg | 莱姆(氏)病 阴性 |
JL-FA-127 | Malaria | 疟疾 |
JL-FA-128 | Mononucleosis (infectious) | 单核细胞增多症（有传染性的） |
JL-FA-129 | Mononucleosis Negative | 单核细胞增多症阴性 |
JL-FA-130 | Measles Negative | 麻疹 阴性 |
JL-FA-131 | Measles IgG | 麻疹 IGG |
JL-FA-132 | Measles IgM | 麻疹 IGM |
JL-FA-133 | Microsomal Anti-thyroid peroxidase antibody (TPO) Positive Plasma Standard Titer (typically 1,000-3,000 IU/mL) | 微粒体抗甲状腺过氧化物酶抗体 |
JL-FA-134 | Microsomal Anti-thyroid peroxidase antibody (TPO) Negative Plasma | 微粒体抗甲状腺过氧化物酶抗体 |
JL-FA-135 | Anti-mitochondrial antibody (AMA) | 抗线粒体抗体 |
JL-FA-136 | Multiple Sclerosis | 多发性硬化症 |
JL-FA-137 | Mumps IgG | 流行性腮腺炎 IGG |
JL-FA-138 | Mumps Ab IgM | 流行性腮腺炎抗体 IGM |
JL-FA-139 | Mumps Antibody Negative Plasma | 流行性腮腺炎抗体阴性血浆 |
JL-FA-140 | Mumps Antibody Negative Serum | 流行性腮腺炎抗体阴性血清 |
JL-FA-141 | Myeloma Plasma | 骨髓瘤血浆 |
JL-FA-142 | Myeloma IgA | 骨髓瘤IGA |
JL-FA-143 | Myeloma IgE | 骨髓瘤IGE |
JL-FA-144 | Myeloma IgG | 骨髓瘤IGG |
JL-FA-145 | Myeloma IgM | 骨髓瘤IGM |
JL-FA-146 | Mycoplasma | 支原体 |
JL-FA-147 | Mycoplasma Negative | 支原体阴性 |
JL-FA-148 | Mycoplasma IgG | 支原体IGG |
JL-FA-149 | Mycoplasma IgM | 支原体IGM |
JL-FA-150 | Mycoplasma PCR | 支原体PCR |
JL-FA-151 | Normal Human Plasma | 正常人血浆 |
JL-FA-152 | Normal Human Serum | 正常人血清 |
JL-FA-153 | Nuclear Antibody Centromere | 核抗体着丝粒 |
JL-FA-154 | Nuclear Antibody, Speckled ANA | 核抗体，斑点抗核抗体 |
JL-FA-155 | Nuclear Antibody, Nucleolar ANA | 核抗体,核仁抗核抗体 |
JL-FA-156 | Nuclear Antibody, Homogeneous ANA | 核抗体，同质抗核抗体 |
JL-FA-157 | Nuclear Antiobody, Speckled. (ANA) Negative | 核抗体，斑点。抗核抗体阴性 |
JL-FA-158 | P-ANCA (associated neutrophil cytoplasmic antibodies) | 相关的嗜中性粒细胞胞浆抗体 |
JL-FA-159 | Parietal Cell Antibody (PCA) | 胃)壁细胞抗体 |
JL-FA-160 | Parvo positive plasma | 细小病毒阳性血浆 |
JL-FA-161 | Parvo IgM | 细小病毒 IGM |
JL-FA-162 | Parvo IgG | 细小病毒 IGG |
JL-FA-163 | Parvo Negative Plasma | 细小病毒阴性血浆 |
JL-FA-164 | Parvo DNA positive | 细小病毒 DNA 阳性 |
JL-FA-165 | Phospholipid Positive Plasma | 磷脂阳性血浆 |
JL-FA-166 | Prothrombin | 凝血酶原,凝血因子 |
JL-FA-167 | Rheumatoid Factor (RF) <1000 IU/mL | 类风湿因子<1000 IU/mL |
JL-FA-168 | Rheumatoid Factor (RF) 1001-2000 IU/mL | 类风湿因子1001-2000 IU/mL |
JL-FA-169 | Rheumatoid Factor (RF) 2001-4000 IU/mL | 类风湿因子 2001-4000 IU/mL |
JL-FA-170 | Rheumatoid Factor (RF) 4001-5000 IU/mL | 类风湿因子 4001-5000 IU/mL |
JL-FA-171 | Rheumatoid Factor (RF) >5000 IU/mL | 类风湿因子>5000 IU/mL |
JL-FA-172 | Ribonucleoprotein (RNP) Positive | 核糖核蛋白阳性 |
JL-FA-173 | Rubella Chimeric | 风疹 |
JL-FA-174 | Rubella Negative | 风疹阴性 |
JL-FA-175 | Rubella IgG | 风疹IGG |
JL-FA-176 | Rubella IgM | 风疹IGM |
JL-FA-177 | Rubeola Negative Plasma | 风疹阴性血浆 |
JL-FA-178 | Rubeola IgG | 风疹IGG |
JL-FA-179 | Scleroderma (Scl-70) Pos | 胶原沉着病,硬皮病,硬皮症 阳性 |
JL-FA-180 | Scleroderma (Scl-70) Negative | 硬皮病阴性 |
JL-FA-181 | Sickle Cell Fresh Whole Blood | 镰刀形红细胞新鲜全血 |
JL-FA-182 | Smith (SM) | 抗Smith抗体阳性血清（SLE的特征性抗体） |
JL-FA-183 | SMITH RNP | 抗RNP抗体阳性血清（SLE的特征性抗体） |
JL-FA-184 | Smooth Muscle (ASMA) | 抗平滑肌抗体阳性血清 |
JL-FA-185 | Sjogren syndrome antigen A (SSA) Positive | 舍格伦综合征或干燥综合征抗原A 阳性 |
JL-FA-186 | Sjogren syndrome antigen B (SSB) Positive | 舍格伦综合征抗原B 阳性 |
JL-FA-187 | Sjogren syndrome antigen B (SSB) Negative | 舍格伦综合征抗原B阴性 |
JL-FA-188 | Streptolysin O Ab (ASO) | 链球菌溶血素O抗体 |
JL-FA-189 | Syphilis (RPR - Rapid Plasma Reagin) Positive Plasma | 梅毒（梅毒-快速血浆反应）阳性血浆 |
JL-FA-190 | Syphilis (RPR - Rapid Plasma Reagin) Negative Plasma | 梅毒（梅毒-快速血浆反应）阴性血浆 |
JL-FA-191 | Syphilis/ATA/T. pallidum IgG | 梅毒ATA/T，苍白球IGG |
JL-FA-192 | Syphilis/ATA/T. pallidum IgM | 梅毒ATA/T，苍白球IGM |
JL-FA-193 | Systemic Lupus Erythematosus (SLE) Positive | 全身性红斑狼疮阳性 |
JL-FA-194 | Systemic Lupus Erythematosus (SLE) Negative | 全身性红斑狼疮阴性 |
JL-FA-195 | TG/TPO Positive (Standard Titer 1,000 - 3000 IU/mL) | 甲状腺球蛋白/甲状腺过氧化物酶阳性 |
JL-FA-196 | TG/TPO Negative | 甲状腺球蛋白/甲状腺过氧化物酶阴性 |
JL-FA-197 | TTG (Tissue Transglutaminase) | 组织转谷氨酰胺酶 |
JL-FA-198 | TTG (Tissue Transglutaminase) IgA | 组织转谷氨酰胺酶 IGA |
JL-FA-199 | ToRCH (Toxo, Rubella, CMV, HSV) Positive | 优生优育（弓形虫，风疹，巨细胞，单胞）阳性 |
JL-FA-200 | ToRCH (Toxo, Rubella, CMV, HSV) Negative | 优生优育（弓形虫，风疹，巨细胞，单胞）阴性 |
JL-FA-201 | Toxoplasmosis (Toxo) | 弓形虫病 |
JL-FA-202 | Toxoplasmosis (Toxo) IgG | 弓形虫病IGG |
JL-FA-203 | Toxoplasmosis (Toxo) IgM | 弓形虫病IGM |
JL-FA-204 | Thyroglobulin (TG) Positive Plasma | 甲状腺球蛋白阳性血浆 |
JL-FA-205 | Thyroglobulin (TG) Negative | 甲状腺球蛋白阴性 |
JL-FA-206 | Varicella-Zoster Virus (VZV) Negative | 水痘-带状疱疹病毒阴性 |
JL-FA-207 | Varicella-Zoster Virus (VZV) IgG | 水痘-带状疱疹病毒IGG |
JL-FA-208 | Varicella-Zoster Virus (VZV) IgM | 水痘-带状疱疹病毒IGM |
JL-FA-209 | West Nile Virus (WNV) | 西尼罗河脑炎病毒 |
JL-FA-210 | West Nile Virus (WNV) IgM | 西尼罗河脑炎病毒IGM |
美国
RTKs通过光激活二聚化作用调节发育是哺乳动物受体的光激活二聚化作用的*个实例。光控制下的RTK提供了强大的光遗传学的方法来启动细胞信号和操纵细胞的行为。新开发的受体引发癌症和血液中内皮细胞复杂的细胞活动。这些细胞代表根据光控行为的新模型，以及能够用新的方法来确定药物。这与癌细胞的相反，细胞信号转导的不受控制的活化导致恶性肿瘤有关的特征，信号转导的光激活可能拯救退行性疾病的细胞存活和功能。
“渐冻症”是运动神经元病的一种，患者逐渐丧失运动机能甚至瘫痪，其中有名的是英国科学家霍金。这种病被认为与神经胶质细胞异常有关，神经胶质细胞可负责维持神经细胞的网络并向神经细胞提供营养。
京都大学教授井上治久率领的研究小组，利用ips细胞制作出可变化为神经胶质细胞的前驱细胞，然后向24只患有渐冻症的实验鼠脊髓各移植了约8万个这种细胞。
结果发现，移植了前驱细胞的24只渐冻症实验鼠的平均生存期为162天，而没有移植的24只实验鼠仅为150天。研究小组说，移植的前驱细胞几乎全部变为神经胶质细胞之一的星形胶质细胞，开始产生维持神经细胞所需的蛋白质，而且这些移植的细胞没有发生癌变。
研究小组还说，实验鼠的10天相当于人类的数个月到半年时间，不过单纯换算为天数比较困难。他们准备今后将这种前驱细胞与利用iPS细胞制作的运动神经细胞一起移植，以调查会取得什么样的效果。
英国布里斯托尔大学，得克萨斯大学西南医学中心和梅奥诊所的研究人员发现，一个基因的突变，削弱了细胞内的正常转运蛋白的能力。
这些蛋白质的缺乏可能是为什么身体会失去负责产生化学物质多巴胺的神经细胞的部分原因。
已知VPS35基因突变与帕金森氏病有关，但直到现在，专家一直无法查明其作用机制。
发表于2014年6月26日的《Current biology》杂志上的这项研究表明，VPS35突变削弱细胞转运货物蛋白质的一个子集到正确目的地的能力。
他们还确定了影响这个突变的三个额外蛋白质，其中的一些与帕金森氏病有关。
布里斯托尔大学的生物化学教授彼得·卡伦说：“与VPS35突变相关的分子缺陷的发现，为研究这种疾病的病因提供了一个令人兴奋的潜在途径。本研究显著提供越来越多的证据表明，VPS35是retromer复合体中的一部分，是帕金森氏病和其他神经退行性疾病的一个潜在治疗靶点。”
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【公司名称】 广州健仑生物科技有限公司
【】 杨永汉
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【公司地址】 广州清华科技园创新基地番禺石楼镇创启路63号二期2幢101-3室
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RTKs regulate development through photoactivation dimerization as the first instance of photoreactive dimerization of mammalian receptors. RTK under light control provides a powerful method of optogenetics to initiate cellular signaling and manipulate cellular behavior. The newly developed receptors trigger complex cellular activities of cancer and endothelial cells in the blood. These cells represent new models based on photocontrol behavior, as well as being able to identify new drugs in new ways. This is in contrast to cancer cells, where uncontrolled activation of cell signaling leads to malignancy-related features, and photo-activation of signal transduction may rescue the cell survival and function of degenerative diseases.
"Asymptomatic" is a kind of motor neuron disease, patients gradually lose motor function or even paralysis, of which the most famous is the British scientist Hawking. The disease is thought to be associated with abnormalities in glial cells, which can be responsible for maintaining the network of nerve cells and providing nutrients to nerve cells.
A team led by Professor Inoue Ishiguro of Kyoto University made ips cells to make precursor cells that could change into glial cells. Then, about 80,000 of these 24 cells were transplanted into the spinal cord of 24 experimental rats with gradual freezing frost.
The results showed that the average survival time of 24 mice with asherogenic syndrome transplanted with precursor cells was 162 days compared to only 150 days with 24 mice without transplantation. The team said almost all of the transplanted precursor cells became astrocytes, one of the glial cells, and started producing the proteins needed to sustain the nerve cells, and none of the transplanted cells were cancerous.
The team also said that 10 days of experimental mice is equivalent to a few months to six months of human life, but simple conversion to days is more difficult. In the future, they plan to transplant such precursor cells together with motor nerve cells made from iPS cells to investigate what effect they can achieve.
Researchers at the University of Bristol, the University of Texas Southwestern Medical Center and Mayo Clinic have found that a mutation in a gene impairs the ability of normal transporters within the cell.
The lack of these proteins may be part of the reason why the body loses nerve cells responsible for the chemical dopamine production.
Known VPS35 gene mutation and Parkinson's disease, but until now, experts have been unable to identify the mechanism of action.
The study, published in Current Biology, June 26, 2014, shows that VPS35 mutations impair the ability of cells to transport a subset of cargo proteins to the correct destination.
They also identified three additional proteins that affect this mutation, some of which are related to Parkinson's disease.
Peter Karen, a professor of biochemistry at the University of Bristol, said: "The discovery of molecular defects associated with mutations in VPS35 provides an exciting and potential pathway for studying the etiology of this disease." This study significantly provided more and more There is much evidence that VPS35 is part of the retromer complex and is a potential therapeutic target for Parkinson's disease and other neurodegenerative diseases. "
