衣原体阴性对照品（Chlamydia Neg）
| 型 号： | 美国Seracare |
| 报 价： |  |
美国Seracare衣原体阴性对照品（Chlamydia Neg） 阳性质控品对照品 需要了解更多Seracare产品可以咨询我们，本产品由广州健仑生物科技有限公司提供
- 产品描述
美国Seracare衣原体阴性对照品（Chlamydia Neg）
广州健仑生物科技有限公司
广州健仑长期供应各种生物原料，主要代理品牌：美国Seracare、西班牙Certest、美国Fuller等等。
主要产品包括各种标准品、阳性对照品、单克隆抗原抗体。
其中常见的有：弓形虫病、西尼罗河病毒、类风湿因子、疟疾、麻疹、莱姆病、百日咳杆菌、大肠杆菌、鼠伤寒沙门氏菌、李斯特菌等阳性对照品。
美国Seracare衣原体阴性对照品（Chlamydia Neg）
我司还提供其它进口或国产试剂盒：登革热、疟疾、流感、A链球菌、合胞病毒、腮病毒、乙脑、寨卡、黄热病、基孔肯雅热、克锥虫病、违禁品滥用、肺炎球菌、军团菌、化妆品检测、食品安全检测等试剂盒以及日本生研细菌分型诊断血清、德国SiFin诊断血清、丹麦SSI诊断血清等产品。
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【Seracare产品介绍】
编号 | 英文名称 | 中文名称 |
JL-FA-01 | Amebiasis (AME) | 阿米巴病 |
JL-FA-02 | Allergens, Rast scores | 过敏原，放射性过敏原吸收实验。指对特定的人群引起免疫反应或者过敏反应的食品中的蛋白质 |
JL-FA-03 | Allergens, Rast scores negative | 过敏原，放射性过敏原吸收实验阴性 |
JL-FA-04 | Anti-cyclic citrullinated peptide Antibody (CCP) Arthritis | 抗环瓜氨酸肽抗体 |
JL-FA-05 | ASCA Saccharomyces Cerevi | 人抗酿酒酵母抗体（ASCA） |
JL-FA-06 | Aspergillis | 麴菌病 |
JL-FA-07 | Beta 2 Glycoprotein | β2糖蛋白 |
JL-FA-08 | Beta 2 Glycoprotein IgM | β2糖蛋白 IGM |
JL-FA-09 | Bordela Pertussis | 百日咳杆菌 |
JL-FA-10 | Bordela Pertussis IgM | 百日咳杆菌 IGM |
JL-FA-11 | C-ANCA | C-抗中性粒细胞胞浆抗体（ANCA) |
JL-FA-12 | Cardiolipin | 心肌磷脂 |
JL-FA-13 | Cardiolipin IgA | 心肌磷脂 IGA |
JL-FA-14 | Cardiolipin IgG | 心肌磷脂 IGG |
JL-FA-15 | Cardiolipin IgM | 心肌磷脂 IGM |
JL-FA-16 | Cerebral Spinal Fluid | 脑脊髓液 |
JL-FA-17 | Chagas | 恰加斯病/南美锥虫 |
JL-FA-18 | Chlamydia | 衣原体 |
JL-FA-19 | Chlamydia IgA | 衣原体IGA |
JL-FA-20 | Chlamydia IgG | 衣原体IGG |
JL-FA-21 | Chlamydia IgM | 衣原体IGM |
JL-FA-22 | Chlamydia Neg | 衣原体阴性 |
JL-FA-23 | Clotting Factor C3 | 凝固因子C3 |
JL-FA-24 | Clotting Factor C4 | 凝固因子C4 |
JL-FA-25 | Coccidiodes | 球孢菌 |
JL-FA-26 | Cytomegalovirus (CMV) Neg | 巨细胞病毒抗体阴性 |
JL-FA-27 | CMV IgG | 巨细胞病毒 IGG阳性 |
JL-FA-28 | CMV IgM VCA | 巨细胞病毒 IGM 阳性 |
JL-FA-29 | C-Reactive Protein (CRP) | C-反应蛋白质 |
JL-FA-30 | Dengue Fever | 登革热 |
JL-FA-31 | Dengue Fever IgM | 登革热 IGM |
JL-FA-32 | DS (Double Stranded) DNA | 双链脱氧核糖核酸 |
JL-FA-33 | EBNA (Epstein-Barr nuclear antigen) IgG | EB病毒核抗原 IGG |
JL-FA-34 | EBNA (Epstein-Barr nuclear antigen) IgM | EB病毒核抗原 IGM |
JL-FA-35 | Epstein Barr Virus (EBV) Negative Plasma | EB病毒阴性血浆 |
JL-FA-36 | Epstein Barr Virus (EBV) EA IgM | EB病毒早期抗原 IGM |
JL-FA-37 | Epstein Barr Virus (EBV) VCA IgM | EB病毒壳蛋白 IGM |
JL-FA-38 | Epstein Barr Virus (EBV) EA IgG | EB病毒早期抗原 IGG |
JL-FA-39 | EMA (Endomysial Antibodies) | 肌内膜 |
JL-FA-40 | Gliadin | 麸蛋白,麦醇溶蛋白,麦胶蛋白 |
JL-FA-41 | Gliadin IgG | 麦醇溶蛋白 IGG |
JL-FA-42 | Gliadin IgA | 麦醇溶蛋白 IGA |
JL-FA-43 | Glomerular Basement Membrane (GBMA) | 肾小球基底膜病 |
JL-FA-44 | Helicobacter pylori IgA | 幽门螺旋杆菌IGA |
JL-FA-45 | Helicobacter pylori IgG | 幽门螺旋杆菌IGG |
JL-FA-46 | Helicobacter pylori IgM | 幽门螺旋杆菌IGM |
JL-FA-47 | Helicobacter pylori Negative | 幽门螺旋杆菌阴性 |
JL-FA-48 | Helicobacter pylori Positive Plasma | 幽门螺旋杆菌阴性血浆 |
JL-FA-49 | Hepatitis A Virus (HAV) Pos. Plasma | 甲型肝炎病毒阳性血浆 |
JL-FA-50 | Hepatitis A Virus (HAV) IgM | 甲型肝炎病毒IGM |
JL-FA-51 | Hepatitis B Core (HBc) IgG | 乙型肝炎病毒核心 IGG |
JL-FA-52 | Hepatitis B Core (HBc) IgM | 乙型肝炎病毒核心 IGM |
JL-FA-53 | Anti Hbe (Antibody to HBV antigen) | 乙肝抗体 |
JL-FA-54 | Hepatitis Delta Virus | 丁型肝炎病毒 |
JL-FA-55 | HBeAg (HBV e antigen) | 乙肝 E抗原 |
JL-FA-56 | anti-HBs (HBV surface antibody) | 乙肝表面抗体 |
JL-FA-57 | Hepatitis B (HBsAg) "Chronic" | 乙型肝炎（乙肝表面抗原）“慢性病 |
JL-FA-58 | HBsAg (HBV surface antigen) Serum | 乙肝表面抗原血清 |
JL-FA-59 | HBsAg (AD) | 乙肝表面抗原（AD） |
JL-FA-60 | HBsAg (AY) | 乙肝表面抗原（AY） |
JL-FA-61 | HBV Positive Plasma | 乙肝阳性血浆 |
JL-FA-62 | HBV DNA Plasma | 乙肝DNA血浆 |
JL-FA-63 | HBV DNA Serum | 乙肝DNA血清 |
JL-FA-64 | HBV DNA type A | A型 乙肝DNA |
JL-FA-65 | HBV DNA type B | B型 乙肝DNA |
JL-FA-66 | HBV DNA type C | C型 乙肝DNA |
JL-FA-67 | HBV DNA type D | D型 乙肝DNA |
JL-FA-68 | HBV DNA type E | E型 乙肝DNA |
JL-FA-69 | HBV DNA type F | F型 乙肝DNA |
JL-FA-70 | HBV Antibody HCV Antibody Plasma CO-INFECTED | 乙肝和丙肝联合感染血浆 |
JL-FA-71 | HCV (Hepatitis C Virus) Antibody | 丙型肝炎抗体 |
JL-FA-72 | HCV Core Antigen Positive | 丙肝核心抗原 阳性 |
JL-FA-73 | HCV RNA PLASMA Genotype 1 | 基因1型丙肝RNA 血浆 |
JL-FA-74 | HCV RNA PLASMA Genotype 2 | 基因2型丙肝RNA 血浆 |
JL-FA-75 | HCV RNA PLASMA Genotype 3 | 基因3型丙肝RNA 血浆 |
JL-FA-76 | HCV RNA PLASMA Genotype 4 | 基因4型丙肝RNA 血浆 |
JL-FA-77 | HCV RNA PLASMA Genotype 5 | 基因5型丙肝RNA 血浆 |
JL-FA-78 | HCV RNA PLASMA Genotype 6 | 基因6型丙肝RNA 血浆 |
JL-FA-79 | HCV Riba single band | 丙肝免疫印迹单波段 |
JL-FA-80 | HCV RIBA Pos. (multiple bands) | 丙肝免疫印迹阳性多波段 |
JL-FA-81 | HCV Negative | 丙肝阴性 |
JL-FA-82 | HCV RNA Pos (quantitative) | 丙肝RNA阳性（定量） |
JL-FA-83 | Hepatitis E | 戊型肝炎 |
JL-FA-84 | Herpes Simplex Virus (HSV)1/2 Positive Plasma | 单纯性疱疹病毒1/2阳性血浆 |
JL-FA-85 | Herpes Simplex Virus (HSV) 1 Negative Plasma | 单纯性疱疹病毒1 阴性血浆 |
JL-FA-86 | Herpes Simplex Virus (HSV) 1 IgG | 单纯性疱疹病毒1 IGG |
JL-FA-87 | Herpes Simplex Virus (HSV 1) IgM | 单纯性疱疹病毒1 IGM |
JL-FA-88 | Herpes Simplex Virus (HSV) 2 IgG | 单纯性疱疹病毒2 IGG |
JL-FA-89 | Herpes Simplex Virus (HSV) 2 IgM | 单纯性疱疹病毒2 IGG |
JL-FA-90 | Histone | 组蛋白 |
JL-FA-91 | Human Anti Mouse Ab (HAMA) | 人抗鼠抗体 |
JL-FA-92 | Human immunodeficiency virus (HIV) 1 Neg | HIV I 阴性 |
JL-FA-93 | anti Human immunodeficiency virus (HIV) 1 Plasma | 抗HIV I 血浆 |
JL-FA-94 | anti Human immunodeficiency virus (HIV) 1 Serum | 抗HIV I 血清 |
JL-FA-95 | anti Human immunodeficiency virus (HIV) 2 Western Blot Tested | 抗HIV 2 免疫印迹 |
JL-FA-96 | anti Human immunodeficiency virus (HIV) 1/2 2 HIV (+) | 抗HIV 1/2 2 HIV阳性 |
JL-FA-97 | Human immunodeficiency virus (HIV) Ag | HIV抗原 |
JL-FA-98 | HIV RNA (quantitative) Plasma | HIV RNA 定量血浆 |
JL-FA-99 | HIV RNA (quantitative) Serum | HIV RNA 定量血清 |
JL-FA-100 | HIV1 Subtype A | HIV1 亚型A |
JL-FA-101 | HIV1 Subtype B | HIV1 亚型B |
JL-FA-102 | HIV1 Subtype C | HIV1 亚型C |
JL-FA-103 | HIV1 Subtype D | HIV1 亚型D |
JL-FA-104 | HIV1 Subtype E | HIV1 亚型E |
JL-FA-105 | HIV1 Subtype F | HIV1 亚型F |
JL-FA-106 | HIV1 Subtype G | HIV1 亚型G |
JL-FA-107 | HIV1 Subtype H | HIV1 亚型H |
JL-FA-108 | HIV1 Subtype J | HIV1 亚型J |
JL-FA-109 | HIV1 Subtype K | HIV1 亚型K |
JL-FA-110 | HIV1 Group O | HIV1 亚型O |
JL-FA-111 | Human immunodeficiency virus (HIV) 2 Antibody Plasma | HIV 2 抗体血浆 |
JL-FA-112 | Human immunodeficiency virus (HIV) 2 Antibody Serum | HIV 2 抗体血清 |
JL-FA-113 | HPV (Human Papiloma Virus) Negative | 人乳状瘤病毒HPV阴性 |
JL-FA-114 | HPV (Human Papiloma Virus) Positive | 人乳状瘤病毒HPV阳性 |
JL-FA-115 | Human immunodeficiency virus (HIV) Antibody HCV Antibody Plasma COINFECTED | HIV 抗体 HCV |
JL-FA-116 | Human T-cell Lymphotropic Virus (HTLV) I/II | 人嗜T淋巴细胞病毒(HTLV) I/II |
JL-FA-117 | Human T-cell Lymphotropic Virus (HTLV) I | 人嗜T淋巴细胞病毒(HTLV) I |
JL-FA-118 | Human T-cell Lymphotropic Virus (HTLV) II | 人嗜T淋巴细胞病毒(HTLV) II |
JL-FA-119 | Jo-1 | 多发性肌炎抗原JO-1 |
JL-FA-120 | IgE < 5,000 Ku/L | IgE < 5,000 Ku/L |
JL-FA-121 | Legionella | 军团杆菌属 |
JL-FA-122 | Leptospira | 军团杆菌属 |
JL-FA-123 | Lyme Disease | 莱姆(氏)病:蜱传播的全身性疾病,常在夏季发生 |
JL-FA-124 | Lyme IgG | 莱姆(氏)病 IGG |
JL-FA-125 | Lyme IgM | 莱姆(氏)病 IGM |
JL-FA-126 | Lyme Disease Neg | 莱姆(氏)病 阴性 |
JL-FA-127 | Malaria | 疟疾 |
JL-FA-128 | Mononucleosis (infectious) | 单核细胞增多症（有传染性的） |
JL-FA-129 | Mononucleosis Negative | 单核细胞增多症阴性 |
JL-FA-130 | Measles Negative | 麻疹 阴性 |
JL-FA-131 | Measles IgG | 麻疹 IGG |
JL-FA-132 | Measles IgM | 麻疹 IGM |
JL-FA-133 | Microsomal Anti-thyroid peroxidase antibody (TPO) Positive Plasma Standard Titer (typically 1,000-3,000 IU/mL) | 微粒体抗甲状腺过氧化物酶抗体 |
JL-FA-134 | Microsomal Anti-thyroid peroxidase antibody (TPO) Negative Plasma | 微粒体抗甲状腺过氧化物酶抗体 |
JL-FA-135 | Anti-mitochondrial antibody (AMA) | 抗线粒体抗体 |
JL-FA-136 | Multiple Sclerosis | 多发性硬化症 |
JL-FA-137 | Mumps IgG | 流行性腮腺炎 IGG |
JL-FA-138 | Mumps Ab IgM | 流行性腮腺炎抗体 IGM |
JL-FA-139 | Mumps Antibody Negative Plasma | 流行性腮腺炎抗体阴性血浆 |
JL-FA-140 | Mumps Antibody Negative Serum | 流行性腮腺炎抗体阴性血清 |
JL-FA-141 | Myeloma Plasma | 骨髓瘤血浆 |
JL-FA-142 | Myeloma IgA | 骨髓瘤IGA |
JL-FA-143 | Myeloma IgE | 骨髓瘤IGE |
JL-FA-144 | Myeloma IgG | 骨髓瘤IGG |
JL-FA-145 | Myeloma IgM | 骨髓瘤IGM |
JL-FA-146 | Mycoplasma | 支原体 |
JL-FA-147 | Mycoplasma Negative | 支原体阴性 |
JL-FA-148 | Mycoplasma IgG | 支原体IGG |
JL-FA-149 | Mycoplasma IgM | 支原体IGM |
JL-FA-150 | Mycoplasma PCR | 支原体PCR |
JL-FA-151 | Normal Human Plasma | 正常人血浆 |
JL-FA-152 | Normal Human Serum | 正常人血清 |
JL-FA-153 | Nuclear Antibody Centromere | 核抗体着丝粒 |
JL-FA-154 | Nuclear Antibody, Speckled ANA | 核抗体，斑点抗核抗体 |
JL-FA-155 | Nuclear Antibody, Nucleolar ANA | 核抗体,核仁抗核抗体 |
JL-FA-156 | Nuclear Antibody, Homogeneous ANA | 核抗体，同质抗核抗体 |
JL-FA-157 | Nuclear Antiobody, Speckled. (ANA) Negative | 核抗体，斑点。抗核抗体阴性 |
JL-FA-158 | P-ANCA (associated neutrophil cytoplasmic antibodies) | 相关的嗜中性粒细胞胞浆抗体 |
JL-FA-159 | Parietal Cell Antibody (PCA) | 胃)壁细胞抗体 |
JL-FA-160 | Parvo positive plasma | 细小病毒阳性血浆 |
JL-FA-161 | Parvo IgM | 细小病毒 IGM |
JL-FA-162 | Parvo IgG | 细小病毒 IGG |
JL-FA-163 | Parvo Negative Plasma | 细小病毒阴性血浆 |
JL-FA-164 | Parvo DNA positive | 细小病毒 DNA 阳性 |
JL-FA-165 | Phospholipid Positive Plasma | 磷脂阳性血浆 |
JL-FA-166 | Prothrombin | 凝血酶原,凝血因子 |
JL-FA-167 | Rheumatoid Factor (RF) <1000 IU/mL | 类风湿因子<1000 IU/mL |
JL-FA-168 | Rheumatoid Factor (RF) 1001-2000 IU/mL | 类风湿因子1001-2000 IU/mL |
JL-FA-169 | Rheumatoid Factor (RF) 2001-4000 IU/mL | 类风湿因子 2001-4000 IU/mL |
JL-FA-170 | Rheumatoid Factor (RF) 4001-5000 IU/mL | 类风湿因子 4001-5000 IU/mL |
JL-FA-171 | Rheumatoid Factor (RF) >5000 IU/mL | 类风湿因子>5000 IU/mL |
JL-FA-172 | Ribonucleoprotein (RNP) Positive | 核糖核蛋白阳性 |
JL-FA-173 | Rubella Chimeric | 风疹 |
JL-FA-174 | Rubella Negative | 风疹阴性 |
JL-FA-175 | Rubella IgG | 风疹IGG |
JL-FA-176 | Rubella IgM | 风疹IGM |
JL-FA-177 | Rubeola Negative Plasma | 风疹阴性血浆 |
JL-FA-178 | Rubeola IgG | 风疹IGG |
JL-FA-179 | Scleroderma (Scl-70) Pos | 胶原沉着病,硬皮病,硬皮症 阳性 |
JL-FA-180 | Scleroderma (Scl-70) Negative | 硬皮病阴性 |
JL-FA-181 | Sickle Cell Fresh Whole Blood | 镰刀形红细胞新鲜全血 |
JL-FA-182 | Smith (SM) | 抗Smith抗体阳性血清（SLE的特征性抗体） |
JL-FA-183 | SMITH RNP | 抗RNP抗体阳性血清（SLE的特征性抗体） |
JL-FA-184 | Smooth Muscle (ASMA) | 抗平滑肌抗体阳性血清 |
JL-FA-185 | Sjogren syndrome antigen A (SSA) Positive | 舍格伦综合征或干燥综合征抗原A 阳性 |
JL-FA-186 | Sjogren syndrome antigen B (SSB) Positive | 舍格伦综合征抗原B 阳性 |
JL-FA-187 | Sjogren syndrome antigen B (SSB) Negative | 舍格伦综合征抗原B阴性 |
JL-FA-188 | Streptolysin O Ab (ASO) | 链球菌溶血素O抗体 |
JL-FA-189 | Syphilis (RPR - Rapid Plasma Reagin) Positive Plasma | 梅毒（梅毒-快速血浆反应）阳性血浆 |
JL-FA-190 | Syphilis (RPR - Rapid Plasma Reagin) Negative Plasma | 梅毒（梅毒-快速血浆反应）阴性血浆 |
JL-FA-191 | Syphilis/ATA/T. pallidum IgG | 梅毒ATA/T，苍白球IGG |
JL-FA-192 | Syphilis/ATA/T. pallidum IgM | 梅毒ATA/T，苍白球IGM |
JL-FA-193 | Systemic Lupus Erythematosus (SLE) Positive | 全身性红斑狼疮阳性 |
JL-FA-194 | Systemic Lupus Erythematosus (SLE) Negative | 全身性红斑狼疮阴性 |
JL-FA-195 | TG/TPO Positive (Standard Titer 1,000 - 3000 IU/mL) | 甲状腺球蛋白/甲状腺过氧化物酶阳性 |
JL-FA-196 | TG/TPO Negative | 甲状腺球蛋白/甲状腺过氧化物酶阴性 |
JL-FA-197 | TTG (Tissue Transglutaminase) | 组织转谷氨酰胺酶 |
JL-FA-198 | TTG (Tissue Transglutaminase) IgA | 组织转谷氨酰胺酶 IGA |
JL-FA-199 | ToRCH (Toxo, Rubella, CMV, HSV) Positive | 优生优育（弓形虫，风疹，巨细胞，单胞）阳性 |
JL-FA-200 | ToRCH (Toxo, Rubella, CMV, HSV) Negative | 优生优育（弓形虫，风疹，巨细胞，单胞）阴性 |
JL-FA-201 | Toxoplasmosis (Toxo) | 弓形虫病 |
JL-FA-202 | Toxoplasmosis (Toxo) IgG | 弓形虫病IGG |
JL-FA-203 | Toxoplasmosis (Toxo) IgM | 弓形虫病IGM |
JL-FA-204 | Thyroglobulin (TG) Positive Plasma | 甲状腺球蛋白阳性血浆 |
JL-FA-205 | Thyroglobulin (TG) Negative | 甲状腺球蛋白阴性 |
JL-FA-206 | Varicella-Zoster Virus (VZV) Negative | 水痘-带状疱疹病毒阴性 |
JL-FA-207 | Varicella-Zoster Virus (VZV) IgG | 水痘-带状疱疹病毒IGG |
JL-FA-208 | Varicella-Zoster Virus (VZV) IgM | 水痘-带状疱疹病毒IGM |
JL-FA-209 | West Nile Virus (WNV) | 西尼罗河脑炎病毒 |
JL-FA-210 | West Nile Virus (WNV) IgM | 西尼罗河脑炎病毒IGM |
美国Seracare
此外研究人员还发现，对那些“真球迷”而言，镜像神经元还有助于他们更好地“读懂”球赛。对比实验显示，在观赛的时候，那些“真球迷”能够更好地预测球路走向，此时，他们的镜像神经元比那些“伪球迷”更活跃。
维也纳医科大学脑研究中心的奥梅拉·瓦伦蒂说：“镜像神经元能使我们凭直觉捕捉到其他人的意图，而且我们可以根据过往的经验更好地理解这些意图或行为。”
研究人员说，对镜像神经元的研究开始于30年前，如今还处于初步阶段。深入研究镜像神经元可以揭示人类社交互动和模仿学习的奥秘，比如，婴儿学习成人的表情就是因为有镜像神经元，而幼儿自闭症可能与镜像神经元功能失调有关，刺激镜像神经元功能还将有助于中风偏瘫病人恢复行动能力。
人类有许多慢性病，像多发性硬化症、I型糖尿病和类风湿性关节炎，是由免疫系统以某种方式“滥杀无辜”引起的，这种免疫系统伤害自身组织和器官的疾病被称为自体免疫疾病。目前，自体免疫疾病尚无法治愈，而现有的多数药物则无法区分正常免疫细胞和会发动自体攻击的免疫细胞，所以它们虽然可暂时缓解症状，但也消灭了抑制有害细菌和病毒所必需的正常免疫细胞，从长期看会让病人更脆弱。
研究负责人、美国国家卫生研究院黏膜免疫研究室主任陈万军对新华社记者说，自体免疫疾病的一个共同的重要发病机理是，患者免疫系统的正常平衡被破坏，导致患者体内充斥着大量炎症细胞和因子。所以理论上zui有效的治疗方法应该是，首先把这个已失去平衡、免疫反应过强的状态打破，然后再重新诱导免疫系统建立一个新的秩序，即恢复正常的平衡状态，让免疫细胞对自己的组织和器官耐受而不是破坏。“简单地说，基本原理就是‘打破一个旧世界，建立一个新世界’”。
陈万军等人采取的新方案是，首先诱导实验鼠的自身免疫细胞凋亡，让这些细胞被体内一种叫做巨噬细胞的吞噬细胞“吃掉”，之后巨噬细胞会产生一种重要的免疫调控抑制因子TGFβ。TGFβ在体内会形成一个免疫调控的微环境，在这种微环境下再注射一种蛋白多肽，去刺激新的未分化的“原始”或“处女”T细胞，这些T细胞在TGFβ占优势的微环境中，会分化成免疫调节T细胞，而不是破坏自身组织和器官的炎症细胞。
他说：“这些免疫调节细胞会进入相应的、有自身免疫病的组织和器官，防止和抑制破坏自身组织的免疫炎症细胞的产生或发挥功能，从而让疾病得到缓解甚至治愈。”
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In addition, researchers also found that, for those "real fans", mirror neurons also help them better "read" the game. Comparative experiments show that those "true fans" are better at predicting the direction of the ball during the match, when their mirror neurons are more active than those "pseudo-fans."
Olmela Valenti at the Brain Research Center at the Medical University of Vienna said: "Mirroring neurons enables us to intuitively capture the intentions of others, and we can better understand these intentions or behaviors based on past experience."
Researchers say the study of mirrored neurons began 30 years ago and is still in its infancy. In-depth study of mirrored neurons can reveal the mysteries of human social interaction and mimic learning. For example, infants learning adult expression because of mirror neurons and autistic children may be related to dysfunction of mirrored neurons, stimulating the function of mirrored neurons Will help stroke patients with hemiplegia to restore mobility.
There are many chronic diseases in humans, such as multiple sclerosis, type I diabetes and rheumatoid arthritis, which are caused by the immune system somehow "killing innocent people" and diseases of the immune system that harm their own tissues and organs are said For autoimmune diseases. At present, autoimmune diseases are not yet curable, and most existing medications do not distinguish between normal immune cells and immune cells that trigger autologous attacks. Although they temporarily relieve symptoms, they also eliminate the need for harmful bacteria and viruses Normal immune cells, in the long run will make the patient more vulnerable.
Chen Wanjun, head of the study and Director of the Mucosal Immunization Laboratory at the National Institutes of Health, told Xinhua that a common and important pathogenesis of autoimmune diseases is the breakdown of the normal balance of the patient's immune system, resulting in the patient being flooded with inflammatory cells And factor. Therefore, in theory, the most effective treatment should be the first to break the balance of the immune response is too strong, and then re-induce the immune system to establish a new order, that is, to restore normal equilibrium, so that immune cells on their own Tissue and organs are tolerant rather than destructive. "Simply put, the basic principle is to 'break an old world and build a new world'".
Chen Wanjun and others to take a new program is to first induce experimental mouse immune cell apoptosis, so that these cells are the body of a phagocyte called macrophage eaten, macrophages will produce an important immune Regulatory inhibitor of TGFβ. TGFβ forms an immunomodulatory microenvironment in the body in which a protein polypeptide is reinjected to stimulate new undifferentiated "virgin" or "virgin" T cells that predominate in TGFβ In the microenvironment, it differentiates into immunoregulatory T cells rather than inflammatory cells that destroy their own tissues and organs.
"These immune-regulating cells get into the corresponding autoimmune-damaged tissues and organs, preventing and inhibiting the production or functioning of immune-inflammatory cells that destroy their tissues, thereby allowing the disease to be relieved or even healed."
