康氏立克次体IgG ELISA检测试剂盒

R. conorii IgG ELISA kit

广州健仑生物科技有限公司

主要用途：用于检测人血清中的康氏立克次体IgG抗体

产品规格：96T/盒

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康氏立克次体IgG ELISA检测试剂盒

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【公司名称】 广州健仑生物科技有限公司
【】    杨永汉 
【】 
【腾讯 】 2042552662
【公司地址】 广州清华科技园创新基地番禺石楼镇创启路63号二期2幢101-3室
【企业文化】

研究人员指出，上皮细胞不仅仅是一道物理屏障，在抵御病毒和细菌的过程中，它为免疫细胞提供支持。
研究人员发现，向肠道添加特定的益生菌菌株——植物乳杆菌，可逆转IL-1β快速减少所引起的损伤，解决炎症，并在几个小时之内加速修复。研究指出了一种有趣的可能性，即利用协同宿主-微生物相互作用来干预早期病毒传播和肠道炎症、减轻HIV感染有关的肠道并发症。
Lauren Hirao说：“了解免疫反应中的参与因子，对于开发新的治疗方法非常重要。弄清这些事件如何发展，能够帮助我们发现干预的zui恰当时机。”
姜黄素是姜黄的活性成分，已被科学研究用于许多类型癌症的治疗。姜黄素已被证明具有癌化学预防作用，能扭转，抑制或阻止癌症发展的能力。
磷酸化负责开启和关闭蛋白质，改变蛋白质的功能和活性。皮层蛋白过度磷酸化活化已与癌症侵略性密切相关。亚利桑那州大学的研究人员用姜黄素处理结肠癌细胞发现，姜黄素能关闭皮层蛋白的活性形式，因此，当皮层蛋白被关闭时，癌细胞失去移动能力，并且不能转移到身体的其他部分。
更具体地说，姜黄素通过与皮层蛋白相互作用，并激活PTPN1酶，“关闭”皮层蛋白。PTPN1酶作为一种磷酸酶，可从皮层蛋白中去除磷酸基团，也即“去磷酸化”过程。而皮层蛋白去磷酸化与结肠癌细胞迁移能力降低相关。
通过确定姜黄素激活酶PTPN1，然后关闭皮层蛋白的活性部位pTyr421，研究人员认为其可以开发成针对癌细胞皮层蛋白的化学预防药物，以防止癌症的转移性。
ALK7受体主要发现在脂肪细胞和参与调控代谢的组织中。有趣的是，ALK7突变小鼠的脂肪积累比携带一个功能版本蛋白质的小鼠还要少。到现在为止，人们还不清楚为什么会这样。

The researchers point out that epithelial cells are more than just a physical barrier, they support immune cells in their defense against viruses and bacteria.
The researchers found that adding a specific probiotic strain, Lactobacillus plantarum, to the intestine reverses the damage caused by a rapid decrease in IL-1β, resolves inflammation and speeds healing within hours. The study points to an interesting possibility of using synergistic host-microbe interactions to intervene in early virus transmission and intestinal inflammation and to reduce the intestinal complications associated with HIV infection.
"Understanding the factors involved in the immune response is important for developing new treatments," explains Lauren Hirao, and figuring out how these events have evolved helps us find the right time to intervene. "
Curcumin, the active ingredient of turmeric, has been scientifically used in the treatment of many types of cancer. Curcumin has been shown to have a chemopreventive effect on cancer that can reverse, inhibit or prevent the development of cancer.
Phosphorylation is responsible for turning proteins on and off, altering the function and activity of the protein. Over-phosphorylation of cortical proteins has been implicated in cancer aggressiveness. Using curcumin to treat colon cancer cells, researchers at the University of Arizona found that curcumin turns off the active form of cortical proteins, so that when cortical proteins are switched off, cancerous cells lose their ability to move and can not move to other parts of the body.
More specifically, curcumin "closes" the cortex protein by interacting with cortical proteins and activating the PTPNl enzyme. The PTPN1 enzyme, a phosphatase that removes phosphate groups from the cortex protein, is also known as the "dephosphorylation" process. Dephosphorylation of cortical proteins is associated with decreased colon cell migration.
By identifying the curcumin-activating enzyme, PTPN1, and then shutting down the active site of cortical protein, pTyr421, researchers believe it could be developed as a chemopreventive drug against cancer cell cortex proteins to prevent cancer metastasis.
ALK7 receptors are mainly found in adipocytes and tissues involved in the regulation of metabolism. Interestingly, ALK7 mutant mice have less fat accumulation than mice that carry a functional version of the protein. Until now, it is unclear why this is so.