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【公司名称】 广州健仑生物科技有限公司
【市场部】    杨永汉

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【腾讯  】 2042552662
【公司地址】 广州清华科技园创新基地番禺石楼镇创启路63号二期2幢101-103室

此次研究中，研究人员通过导入这个基因的拷贝，实现了该受体的过量表达。而实际生活中，并不能将这一做法应用到人身上，因而，研究人员将进一步开发靶向这一基因的药物。
一项来自美国俄亥俄州立大学综合癌症中心的证据显示，经基因修饰的免疫细胞或对多发性骨髓瘤的治疗有效果，且这些细胞可在实验室中培养增殖。该研究成果5月28日刊载在Clinical Cancer Research杂志。
研究人员对人类免疫细胞(来自患者的细胞系和骨髓瘤细胞)——T淋巴细胞进行修饰，靶向存在于大多数(占比95%以上)的骨髓瘤细胞中的CS1分子，以期杀死细胞。研究人员在实验室中培养增殖这些修饰细胞，然后将其注入动物模型中，杀死人骨髓瘤细胞。
与对照组t细胞相比，修饰T细胞可以更好地识别过表达CS1的多发性骨髓瘤细胞，并激活。注射修饰T细胞的所有小鼠在接受治疗后存活了44天，而在另外两个对照组中，这一数据只有29%和17%。
这种疗法的一个重要的优势是，这些治疗T细胞在体内有复制潜能，所以或许可以抑制肿瘤生长，长时间内避免复发。
墨尔本的研究人员证明，A型白血病能成功地‘逆转’通过诱骗癌症细胞回到正常发育状态。这一发现使用B-前体急性淋巴细胞白血病(B-ALL)模型，是一种zui常见的影响儿童的癌症。研究人员揭示关闭一个称为Pax5的基因在B-ALL模型中能够诱发癌症，而恢复它的功能能够‘治愈’这种疾病。研究发表在《Genes & Development》期刊上。
研究者之一的刘女士说团队利用一种发展的‘基因开关’技术去抑制然后再活化Pax5在白血病模型中。

In this study, researchers introduced an overexpression of this gene by introducing a copy of this gene. And in real life, this practice can not be applied to people, so the researchers will further develop drugs that target this gene.
Evidence from the Ohio State University Comprehensive Cancer Center shows that genetically modified immune cells or treatments for multiple myeloma can be effective and that these cells can be cultured and proliferated in the laboratory. The research was published May 28 in Clinical Cancer Research.
Researchers have modified human immune cells (cell lines from patients and myeloma cells), T-lymphocytes, to target CS1 molecules present in the majority (over 95%) of myeloma cells in order to kill cell. The researchers cultured and cultured these modified cells in a laboratory and injected them into animal models to kill human myeloma cells.
Modification of T cells allows better identification and activation of CS1-overexpressing multiple myeloma cells as compared to control t-cells. All mice injected with modified T cells survived for 44 days after treatment, compared with only 29% and 17% of the other two controls.
An important advantage of this therapies is that these therapeutic T cells have the potential to replicate in the body and so may be able to inhibit tumor growth and avoid relapse for long periods of time.
Researchers in Melbourne have demonstrated that type A leukemia can successfully 'reverse' by inducing cancer cells to return to their normal developmental state. This finding, using the B-ALL acute lymphoblastic leukemia (B-ALL) model, is one of the most common cancers in children. Researchers have revealed that the closure of a gene called Pax5 is capable of inducing cancer in the B-ALL model, and restoring its function to 'cure' the disease. Research published in "Genes & Development" journal.
Ms. Liu, one of the researchers, said the team took advantage of a newly developed 'gene switch' technique to repress and then reactivate Pax5 in leukemia models.