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【公司名称】 广州健仑生物科技有限公司
【市场部】    杨永汉

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【腾讯  】 2042552662
【公司地址】 广州清华科技园创新基地番禺石楼镇创启路63号二期2幢101-103室

Dickins博士说强制B-ALL 细胞重新开始它们的正常发育能够为治疗白血病提供一种新策略。“然而B-ALL与其它癌症相比相对容易预测，目前的治疗可以持续多年而且具有重大副作用。通过了解特殊的基因改变如何驱动B-ALL，有可能产生行动更快且较少有副作用的特异疗法。”
然而Dickins博士说肿瘤细胞中的基因缺失不是传统上的药物靶点。“癌症发育期间通过开发药物恢复丢失基因的功能是非常困难的，”Dickins博士说。“然而通过了解Pax5缺失引起白血病的机制，我们就可以开始考虑开发药物的方式，能够像恢复Pax5功能一样的效果。基因开关技术用于学习Pax5也能用于了解其它癌症中的‘肿瘤抑制’基因。”
美国亚利桑那州费城蒂格尼提健康巴罗神经学研究所和美国加州大学圣地亚哥分校医学院的研究人员进行的研究显示，人类大脑会在海马体里存储情景记忆，将每一段回忆交付给*的单个细胞，这一发现证实了之前神经计算理论学家长久怀疑的问题。
这个人类神经元显示了针对特定刺激而作出回应的肌动蛋白的形成。

Dr. Dickins said forcing B-ALL cells to restart their normal development could provide a new strategy for the treatment of leukemia. However, B-ALL is relatively easy to predict when compared to other cancers, and the current treatment can last many years with significant side effects. By understanding how specific genetic changes drive B-ALL, it is possible to produce actions that are faster and have fewer side effects therapy."
However, Dr. Dickins said that gene deletion in tumor cells is not a traditional drug target. "It is very difficult to recover lost genes by developing drugs during cancer development," said Dr. Dickins. "However, by understanding the mechanism by which Pax5 deficiency causes leukemia, we can begin to think about ways to develop drugs that have the same effect as Pax5 functions." Gene switch technology for studying Pax5 can also be used to understand 'tumor suppression' in other cancers gene."
A recent study by researchers at the Diaghini Institute of Health Barlow Neurology in Philadelphia, Arizona, and the University of California San Diego School of Medicine found that the human brain stores episodic memories in the hippocampus, delivering each piece of memory to a unique This finding confirms the longstanding suspicion of neuro-computing theorists.
This human neuron shows the formation of actin that responds to a specific stimulus.