韩国SD疟原虫全血检测试纸条

广州健仑生物科技有限公司

（广州健仑生物科技有限公司是集研制开发、销售、服务于一体的优良企业,公司产品涉及临床快速诊断试剂、食品安全检测试剂，违禁品快速检测，动物疾病防疫检测试剂，免疫诊断试剂、临床血液学和体液学检验试剂、微生物检验试剂、分子生物学检验试剂、临床生化试剂、有机试剂等众多领域，同时核心代理Panbio、FOCUS、Qiagen、IBL、CORTEZ、Fuller、Inbios、BinaxNOW、LumuQuick、日本富士、日本生研等多家有名诊断产品集团公司产品，致力于为商检单位、疾病预防控制中心、海关出入境检疫局、卫生防疫单位,缉毒系统，戒毒中心，检验检疫单位、生化企业、科研院所、医疗机构等机构与行业提供*、高品质的产品服务。此外，本公司还开展食品、卫生、环境、药品等多方面的第三方检测服务。）

韩国SD疟原虫全血检测试纸条? 本试剂盒主要是采用胶体金层析的原理制成，用于检测人体血清/血浆/全血标本中，感染的疟原虫抗体，包括了恶性疟原虫和间日疟原虫、卵形疟原虫、三日疟原虫共有抗原的鉴别性检测。

人群易感性 人群对疟疾普遍易感，感染后虽有一定的免疫力，但不持久，各型疟疾之间亦无交叉免疫性，经反复多次感染后，再感染时症状可较轻，甚至无症状，而一般非流行区来的外来人员常较易感染，且症状较重。
People susceptible to the crowd generally susceptible to malaria, although the infection after a certain degree of immunity, but not lasting, there is no cross-immunity between malaria, after repeated infections, re-infection symptoms may be lighter, or even Asymptomatic, while the non-endemic areas of non-migrant workers are often more susceptible to infection, and the symptoms are severe.

：

1 撕开检测卡铝箔袋，取出袋内金标卡。注意：不要让袋内材料暴露于高温高湿环境，撕开铝箔袋后尽快使用。

2将金标卡平放在台面上；并将病人名字和编号写在标签上。

3 取5微升(吸管*刻度处)全血标本，垂直加入金标卡上“加样孔A”内。

4 掰断裂解液瓶子盖子上方的绿色圆头，在“样品孔B”上垂直滴加4滴裂解液。

5 在十五分钟内出结果。注意：必须在15分钟内判读结果，如超时判断，结果无效。

6 请遵循相关法规，妥善处理样本及废弃材料。

7 存储条件：2-30℃；

8 保质期：18个月；

【病原学检测】

疟疾检测,用于检测出虐疾的病原体——疟原虫，是明确诊断的zui直接证据。目前常用的层析法，具有操作简单、灵敏度高和可鉴别虫种等优点，广泛用于疟疾的病原学诊断，是目前zui常用的方法之一。

我司为美国NOVABIOS公司在中国地区战略合作伙伴，负责该公司产品的总经销及售后服务工作。还与各疾控中心，疾病防御中心有合作关系，例如中国疾病预防控制中心 、浙江省疾病预防控制中心  ，详情可以我司工作人员。

（  MOB：杨永汉）  

我司还提供其它进口或国产试剂盒：登革热、疟疾、流感、A链球菌、合胞病毒、腮病毒、乙脑、寨卡、黄热病、基孔肯雅热、克锥虫病、违禁品滥用、肺炎球菌、军团菌、化妆品检测、食品安全检测等试剂盒以及日本生研细菌分型诊断血清、德国SiFin诊断血清、丹麦SSI诊断血清等产品。

广州健仑生物长期供应各种违禁品检测试纸、违禁品检测卡、违禁品检测试剂盒、药筛试纸、药筛试剂盒、吗啡检测试剂盒、巴比妥检测试剂盒等。

想了解更多的产品及服务请扫描下方二维码：

【公司名称】 广州健仑生物科技有限公司
【市场部】    杨永汉

【】 
【腾讯  】 
【公司地址】 广州清华科技园创新基地番禺石楼镇创启路63号二期2幢101-103

多见于老年人，常与季节有关，皮损为继发性。
5.原发性皮肤淀粉样变
皮损呈高粱至绿豆大棕褐色坚硬丘疹，有时皮疹沿皮纹呈念珠状

排列，组织病理上淀粉样蛋白沉积具有特征性改变。扁桃体炎可

分为急性扁桃体炎和慢性扁桃体炎?；技毙源静。ㄈ缧珊烊取?/p>

麻疹、流感、白喉等）后，可引起慢性扁桃体炎，鼻腔有鼻窦感

染也可伴发本病。病源菌以链球菌及葡萄球菌等zui常见。临床表

现为经常咽部不适，异物感，发干、痒，刺激性咳嗽，口臭等症

状。胆囊炎是较常见的疾病，发病率较高。根据其临床表现和临

床经过，又可分为急性和慢性两种类型，常与胆石症合并存在。

右上腹剧痛或绞痛，多见于结石或寄生虫嵌顿梗阻胆囊颈部所致

的急性胆囊炎，疼痛常突然发作，十分剧烈，或呈绞痛样。胆囊

管非梗阻性急性胆囊炎时，右上腹疼痛一般不剧烈，多为持续性

胀痛，随着胆囊炎症的进展，疼痛亦可加重，疼痛呈放射性，zui

常见的放射部位是右肩部和右肩胛骨下角等处。胆囊内结石突然

梗阻或嵌顿胆囊管导致急性胆囊炎，胆囊管扭转、狭窄和胆道蛔

虫或胆道肿瘤阻塞也可引起急性胆囊炎。此外，增龄老化过程中

，胆囊壁逐渐变得肥厚或萎缩，收缩功能减退，造成胆汁淤滞、

浓缩并形成胆酸盐；胆总管末端及Oddi括约肌变得松弛，容易发

生逆行性感染；全身动脉粥样硬化，血液粘滞度增加可加重胆囊

动脉缺血。胆囊管或胆囊颈梗阻后，胆囊内淤滞的胆汁浓缩形成

胆酸盐，后者刺激胆囊黏膜引起化学性胆囊炎（早期）；与此同

时胆汁潴留使胆囊内压力不断增高，膨胀的胆囊首先影响胆囊壁

的静脉和淋巴回流，胆囊出现充血水肿，当胆囊内压>5.39kPa（

55cmH2O）时，胆囊壁动脉血流阻断，胆囊发生缺血性损伤，缺血

的胆囊容易继发细菌感染，加重胆囊炎进程，zui终并发胆囊坏疽

或穿孔。若胆囊管梗阻而没有胆囊壁的血液循环障碍和细菌感染

，则发展为胆囊积液。近年的研究表明，磷脂酶A可因胆汁淤滞或

结石嵌顿从损伤的胆囊黏膜上皮释放，使胆汁中卵磷脂水解成溶

血卵磷脂，后者进而使黏膜上皮细胞的完整性发生变化引起急性

胆囊炎。急性结石性胆囊炎的临床表现和急性无结石性胆囊炎基

本相同。More common in the elderly, often associated with the seasons, skin lesions secondary.
5. Primary cutaneous amyloidosis
Lesions were sorghum to mung bean big brown hard papules, sometimes rashes were rosary beads

Permutations, histopathologically, amyloid deposits have characteristic changes. Tonsillitis can be

Divided into acute tonsillitis and chronic tonsillitis. Acute infectious diseases (such as scarlet fever,

Measles, flu, diphtheria, etc.), can cause chronic tonsillitis, nasal sinus feel

Dye can also be associated with the disease. Pathogens to Streptococcus and Staphylococcus are the most common. Clinical Table

Now often throat discomfort, foreign body sensation, hair dry, itchy, irritating cough, bad breath embolism

shape. Cholecystitis is a more common disease, with a higher incidence. According to its clinical manifestations and immediate

Bed after, can be divided into two types of acute and chronic, often combined with cholelithiasis.

Right upper quadrant pain or colic, more common in stones or parasite obstruction caused by gallbladder neck

Acute cholecystitis, often sudden onset of pain, very intense, or showed cramps. gallbladder

Non-obstructive acute cholecystitis, right upper quadrant pain is not usually intense, mostly persistent

Pain, with the progress of cholecystitis, the pain can be aggravated, the pain was radioactive, most

Common parts of the radiation is the right shoulder and right shoulder blade angle and other places. Gallbladder stones suddenly

Obstruction or incarceration of the cystic duct leads to acute cholecystitis, cystic duct torsion, stenosis and biliary cirrhosis

Obstruction of worms or biliary tumors can also cause acute cholecystitis. In addition, aging process

, Gallbladder wall gradually become hypertrophy or atrophy, systolic dysfunction, resulting in cholestasis,

Concentration and the formation of cholate; common bile duct sphincter and Oddi become loose, easy to hair

Health retrograde infection; systemic atherosclerosis, increased blood viscosity can aggravate the gallbladder

Arterial ischemia. Gallbladder or gallbladder neck obstruction, the formation of gallbladder stasis bile concentration

Cholic acid salt, which stimulates the gallbladder mucosa caused by chemical cholecystitis (early); the same

When bile retention to the gallbladder pressure continues to increase, the first expansion of the gallbladder wall gallbladder

Of venous and lymphatic drainage, congestion and edema of the gallbladder, when the gallbladder pressure> 5.39kPa (

55cmH2O), the gallbladder wall artery blood flow occlusion, ischemic injury of the gallbladder, ischemia

Gallbladder easily secondary to bacterial infection, aggravating cholecystitis process, the ultimate complication of gallbladder gangrene

Or perforated. If the cystic duct obstruction without gallbladder wall blood circulation disorders and bacterial infections

, Then develop into gallbladder fluid. Recent studies have shown that phospholipase A may be due to cholestasis or

Stones incarcerated from the injury of the gallbladder mucosal epithelium release, the bile lecithin hydrolysis

Blood lecithin, which in turn causes changes in the integrity of mucosal epithelial cells caused by acute

cholecystitis. Acute calculous cholecystitis clinical manifestations and acute calculi-based

The same.