嗜肺军团菌(1—7)IgG试剂盒（酶联免疫法）

广州健仑生物科技有限公司

（广州健仑生物科技有限公司是集研制开发、销售、服务于一体的优良企业,公司产品涉及临床快速诊断试剂、食品安全检测试剂，违禁品快速检测，动物疾病防疫检测试剂，免疫诊断试剂、临床血液学和体液学检验试剂、微生物检验试剂、分子生物学检验试剂、临床生化试剂、有机试剂等众多领域，同时核心代理Panbio、FOCUS、Qiagen、IBL、CORTEZ、Fuller、Inbios、BinaxNOW、LumuQuick、日本富士、日本生研等多家有名诊断产品集团公司产品，致力于为商检单位、疾病预防控制中心、海关出入境检疫局、卫生防疫单位,缉毒系统，戒毒中心，检验检疫单位、生化企业、科研院所、医疗机构等机构与行业提供*、高品质的产品服务。此外，本公司还开展食品、卫生、环境、药品等多方面的第三方检测服务。）

主要用途：用于检测尿样中嗜肺军团菌血清型1抗原，以支持军团菌感染的诊断。

产品规格：20T/盒

存储条件：2-30℃

嗜肺军团菌(1—7)IgG试剂盒（酶联免疫法）

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【产品介绍】

嗜肺军团菌(1—7)IgG试剂盒（酶联免疫法）

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【公司名称】 广州健仑生物科技有限公司
【】    杨永汉 
【】 
【腾讯 】 2042552662
【公司地址】 广州清华科技园创新基地番禺石楼镇创启路63号二期2幢101-3室
【企业文化】

库鲁病是zui早被研究的人类朊毒体病，曾经仅见于巴布亚—新几内亚东部高地有食用已故亲人脏器习俗的土著部落，自从这一习俗被废止后已无新发病例。库鲁病潜伏期长，自4—30年不等，起病隐匿，前驱期患者仅感头痛及关节疼痛，继之出现共济病毒调、震颤、不自主运动，后者包括舞蹈症、肌阵挛等，在病程晚期出现进行性加重的痴呆，神经异常。先后有震颤及共济病毒调后有痴呆是本病的临床特征。患者多在起病3～6个月内死亡。
英文名称 Kuru disease1.传染源
感染朊毒体病的动物和人是本病的传染源。
2.传播途径
本病的传播途径尚不十分清楚，但也已证明的途径有：
（1）消病毒道传播 进食朊毒体感染宿主的或加工物可导致感染本病。如库鲁病就是由于巴布亚—新及内亚东部高地的土著部落有食用已故亲人的脏器以示怀念的传播习俗而致该病在当地传播，而牛海绵状脑病，又称疯牛病，是病毒使用加工不当的动物内脏作饲料而致该病在动物中传播。
（2）医源性传播 病毒病毒、注射尸体来源的人体激素等已被证明可引起克—雅病的医源性传播。
小脑性共济病毒调逐渐加重，其中zui突出的为寒战样震颤，至后期痴呆，瘫痪和尿病毒禁。zui终病毒丧病毒运动能力而死亡。病程一般3～9月，通常可分为3个阶段：
zui初阶段：病人感到头疼和关节疼。躯干震颤、步态蹒跚和共济病毒调引起姿势不稳。患者主观感觉站立和步态不稳、往往还自觉发声，手和眼的运动有异常。说话含糊不清，并逐渐加重。病初常常呈现内斜视，并持续存在。运动病毒调先发于下肢而后逐渐累及上肢。站立时为维持平衡，脚趾用力抓住地面。病初即不能以一只脚站立几秒钟，这对诊断本病很有价值。
中期阶段：数周之后出现行走困难，并伴随着肢体颤抖。已不能行。肢体的僵病毒常进一步发展，伴有广泛性的阵挛或不时发生休克样的肌肉不自主运动，偶尔出现手足徐动症和舞蹈病样运动。在突然受噪声或强光刺激时也会发作。通常有踝阵挛，膝阵挛也常见。虽然肌肉活动已很少，但无束状的或真正的衰弱和肌肉萎缩。病人时常情绪不稳。
Kuru is the first to be studied in human prion disease, once found only in Papua - New Guinea Eastern Highlands indigenous tribal custom of eating deceased loved ones organs, since this practice was abolished no longer new cases. Kuru disease has a long incubation period, ranging from 4 to 30 years, with insidious onset. The prodromal patients are only suffering from headache and joint pain, followed by atherosclerosis, tremor and involuntary movements. The latter include chorea, Twin, etc., late in the course of progressive aggravated dementia, neurological abnormalities. There have been tremor and athemia adjusted dementia is the clinical features of the disease. Most patients died within 3 to 6 months of onset.
English name Kuru disease1. Source of infection
Animals infected with prion diseases and people are the source of infection of the disease.
2. The route of transmission
The route of transmission of the disease is not yet very clear, but also proven ways:
(1) Decommissioning of infectious pathways Infectious prion infections Host or processed foods can cause infection. Such as kuru is because Papua - indigenous tribes and eastern highlands of New Guinea have eaten deceased loved ones organs to show the spread of practices caused the miss at the local spread of the disease, and bovine spongiform encephalopathy, also known as mad cow disease, The virus uses improperly processed animal offal for feed and causes the disease to spread in animals.
(2) iatrogenic viruses and human cadaver-derived human hormones have been shown to cause iatrogenic transmission of Creutzfeldt-Jakob disease.
The cerebellar atherosclerotic virus gradually increased, of which the most prominent chills-like tremor, to the late dementia, paralysis and urine virus ban. The virus eventually died of virus movement capacity. The general course of 3 to 9 months, usually divided into three stages:
The initial stage: the patient feels a headache and a joint pain. Torso tremor, staggering gait, and jaundice caused by instability. Subjective feelings of patients standing and gait instability, often also consciously voice, hand and eye movements are abnormal. Ambiguous and gradually aggravating. Esotropia often present early, and persist. The motility virus is first developed in the lower extremities and then gradually in the upper extremities. To maintain balance when standing, toes firmly grasp the ground. Early illness that can not stand for a few seconds with one foot, which is valuable to the diagnosis of the disease.
Mid-term: Walking difficulties occurred weeks later accompanied by limb tremor. Can not do it. The body's stiff virus is often further developed, with extensive clonus or from time to time shock-like muscle involuntary movements, and occasionally appear athetosis and chorea-like movement. It can also occur when suddenly stimulated by noise or light. Usually ankle clonus, knee clonus is also common. Although muscle activity is minimal, there is no bunched or true weakness and muscle atrophy. Patients often emotional instability.