英国进口沙门氏G多价血清鉴定H抗原

广州健仑生物科技有限公司

我司长期供应尼古丁（可替宁）检测试剂盒，违禁品检测试剂盒，单卡检测，3联卡到12联卡，可以自由组合，根据您的需求自由组合，*，性价比高，产品质量很好。

保存要求：除了有特殊说明，免疫检测产品应保存在2-8°C

产品规格：2ml/瓶

保质期：2年

本试剂盒主要用于对病菌细菌进行检测，利用快速玻片凝集检测技术

利用快速玻片凝集和对流免疫电泳(CIE)鉴定流感嗜血杆菌

英国进口沙门氏G多价血清鉴定H抗原

2ml沙门氏菌H抗原(E)多价eh,enx,enzl5血清

沙门氏菌诊断血清H抗原 G（O多价O单价）

沙门氏菌诊断血清H抗原 G（O多价O单价）

【沙门氏知识点】

沙门氏等在霍乱流行时分离到猪霍乱沙门氏菌，故定名为沙门氏菌属。沙门氏菌属有的专对人类致病，有的只对动物致病，也有对人和动物都致病。沙门氏菌病是指由各种类型沙门氏菌所引起的对人类、家畜以及野生不同形式的总称。感染沙门氏菌的人或带菌者的粪便污染食品，可使人发生食物中毒。据统计在世界各国的种类细菌性食物中毒中，沙门氏菌引起的食物中毒常列。我国内陆地区也以沙门氏菌为*。
沙门氏菌病的病原体。属肠杆菌科，革兰氏阴性肠道杆菌。已发现的近一千种（或菌株）。按其抗原成分，可分为甲、乙、丙、丁、戊等基本菌组。其中与人体疾病有关的主要有甲组的副伤寒甲杆菌，乙组的副伤寒乙杆菌和鼠伤寒杆菌，丙组的副伤寒丙杆菌和猪霍乱杆菌，丁组的伤寒杆菌和肠炎杆菌等。除伤寒杆菌、副伤寒甲杆菌和副伤寒乙杆菌引起人类的疾病外，大多数仅能目引起家畜、鼠类和禽类等动物的疾病，但有时也可污染人类的食物而引起食物中毒。
沙门氏菌在水中不易繁殖，但可生存2-3周，冰箱中可生存3-4个月，在自然环境的粪便中可存活1-2个月。沙门氏菌zui适繁殖温度为37℃，在20℃以上即能大量繁殖，因此，低温储存食品是一项重要预防措施。
沙门氏菌是一种常见的食源性致病菌。沙门氏菌鉴定的传统方法主要是根据形态学特征、培养特征、生理生化特征、抗原特征、噬菌体特征等。

我司还有很多种血清学诊断血清、血液检测、免疫检测产品、毒素检测、凝集检测、酶免检测、层析检测、免疫荧光检测产品，。

（ MOB：杨永汉）

我司还提供其它进口或国产试剂盒：登革热、疟疾、流感、A链球菌、合胞病毒、腮病毒、乙脑、寨卡、黄热病、基孔肯雅热、克锥虫病、违禁品滥用、肺炎球菌、军团菌、化妆品检测、食品安全检测等试剂盒以及日本生研细菌分型诊断血清、德国SiFin诊断血清、丹麦SSI诊断血清等产品。

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【公司名称】 广州健仑生物科技有限公司
【市场部】    杨永汉

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【腾讯  】 
【公司地址】 广州清华科技园创新基地番禺石楼镇创启路63号二期2幢101-103

Due to gene defects, the body can lack a certain hydrolase in lysosomes, resulting in the degradation of the corresponding substances and accumulation in the lysosomes, resulting in cell metabolism barriers and the formation of lysosomal storage diseases. The main pathological manifestations are lysosomal overload in organs (liver, kidney, myocardium, skeletal muscles), ie excessive intake of cells or indigestible substances, or decreased activity of lysosomal enzymes, and the age of the organism. Growth, resulting in a large number of lysosomal accumulation in the cell caused by overload. At present, there are known more than one such disease, and there are many kinds of detectable diseases in the country (see entry: lysosomal storage disease). Among them, type II glycogen storage disease was first discovered. Due to a gene defect in the autosomal hepatocyte, the lack of α-glucosidase in the lysosome leads to the inability of glycogen to degrade to glucose, resulting in a large accumulation of glycogen in the liver and muscle. The disease mostly occurs in infants. Clinical manifestations of muscle weakness, heart enlargement, progressive heart failure, more than two years before death, it is also known as heart-type glycogen deposition disease. The cause of rheumatoid arthritis in rheumatoid arthritis is unclear, but the inflammatory changes in the joint periosteal tissue shown by this disease and the erosion of articular chondrocytes are thought to be due to the local release of lysosomes in cells. The reason may be due to a rheumatoid factor, such as anti-IgG, phagocytosed by macrophages, neutrophils, etc., to promote lysosomes escape. Some of these enzymes, such as collagenase, can erode cartilage and produce localized joint damage, while cartilage-digested metabolites, such as chondroitin sulfate, can also contribute to the production of kinins and participate in joint inflammation. During the shock process, the body's microcirculation is disordered, and tissue ischemia and hypoxia affect the energy supply system, making the membrane unstable, causing leakage of lysosomal enzymes, and causing cell and body damage.